Make your own free website on


F.E.A.T. - Chattanooga          NEWSLETTER                     Page 2     


FEAT-Chattanooga Now Listed  Under IGIVE.COM

When doing your on-line Holiday Shopping this season,  remember to purchase through and FEAT-Chattanooga will receive a percentage of the sales.   
WHAT IS IGIVE.COM: is the first and largest network of members, worthy causes, online merchants, and Web sites, brought together to enable shoppers to list and support their favorite worthy causes through online shopping.
     Charitably-minded individuals  --Become members.   
(It’s free!  Always!) Select FEAT Chattanooga as your worthy cause to support. –Shop the mall at
--Raise money for FEAT Chattanooga.
iGiv sends m
onthly checks to all verified causes after they receive payment from affiliated merchants, 120 days in arrears. The minimum for check issuance is $10. 
If our cause has raised less than $10, our accrued funds will be rolled over to the next pay period., inc., 1740 Ridge Ave., Evanston, IL  60201
The fine print from 
     This e-mail is sent to worthy causes listed at our site by members.  Every time our members shop at stores in the Mall at,  a percentage of their purchases can be donated to their favorite worthy causes.  We offer a wide range of quality products from top merchants – from CDs to books, office supplies, flowers,  toys, and videos.  
The causes listed at are controlled by our members.  They add their favorite causes to our system, we do not.  An listing is not an endorsement of by your cause, nor an endorsement of your cause by


Researchers Disover Autism Gene

WASHINGTON (AP) - Scientists have long theorized that about 16 different genes play in who is born with the severe brain disorder autism - and now they've finally found one of those genes.
   A study of 57 autism patients found that 40 percent carry a mutated version of the HOXA1 gene, which plays a
crucial role in early brain development. University of Rochester scientists reported Monday.
Children need to inherit just one copy of the mutated gene from one parent to have autism. In fact, scientists found only one patient, a very severe case, who inherited a copy of the bad gene from both parents, suggesting that when that first happens the fetus usually dies, said lead researcher Patircia Rodier, who heads the university's National Institutes of Health - funded autism research center.
The NIH called the finding a signigicant step in understanding what predisposes people to developing autism. More than 400,000 Americans have the brain disorder, characterized by profound social withdrawal, repetitive behavior and inability to communicate. Research suggests it's caused when something goes wrong during critical fetal brain development - a theory the gene discovery, in the December issue of the journal Teratology, supports.
HOXA1 is one of a family of genese vital to early embryo development because genes in the group turn on or off other genes. HOXA1's specific role is in brain development. Mice who lack this gene have brainstem damage, malformed ears and other classic signs of autism - one reason Rodier's research team decided to check the gene's role in people.
It's not the kind of gene that could ever be fixed with gene therapy. But the discovery may help doctors unravel just how the brain changes when HOXA1 is abnormal, Rodier said.
"If you figure out the brain changes, you're on your way, we hope, to finding better treatments," she said.

On the Net:
NIH autism site:
Teratology Society site with link to study abstract:




Page 1